TEC alleviate CLD by activating PPARγ, and its molecular mechanisms are mainly as follows: 1) restrain macrophage activation and its derived inflammatory factor, such as IL-1β; 2) upregulated the expression of FXR and LXR as well as FXR-regulated and LXR-regulated genes through activating PPARγ to block NFκb-p65 mediated inhibition of FXR and LXR in hepatocyte; 3) enhanced the PPARγ binding to Bsep promoter and increased its expression.