Figure legend
Figure 1. The activation map of two patients with symptomatic
premature ventricular complex originated from the left ventricle summit.
Figure (A) and (B) showed one patient with premature ventricular complex
(PVC) with QRS duration 142ms. Figure (B) showed the earliest activation
site was localized over the distal great cardiac vein (GCV) (EAT: 29 ms
pre-QRS), while in Figure (A) the earliest activation within the left
ventricle (LV) was located at LV summit region over left ventricular
outflow tract (LVOT), just opposite to the EAT of the distal GCV (EAT:
22 ms pre-QRS). The AEAD was 7 (=∣29-22∣) ms. The patient pre-procedural
LV ejection fraction (LVEF) was 55%, and it was 61% after the
procedure. Figure (C) and (D) showed another patient with PVC with QRS
duration 174ms. Figure (D) showed the earliest activation site was
originated from the distal GCV (EAT: 35 ms pre-QRS), while in Figure
(D), the earliest activation site within the LV endocardium was located
near LV summit region over LVOT (EAT: 20ms pre-QRS). The AEAD was 15
(=∣35-20∣) ms. The patient pre-procedural LVEF was 36%, and it became
43% after the ablation procedure. LV, left ventricle; RVOT, right
ventricular outflow tract.
Figure 2. The changes of the PVC burden and the LVEF before and
after ablation. Panel A showed the changes of the PVC burden before and
after the ablation in those with or without PVC induced cardiomyopathy.
Panel B showed changes of LVEF before and after ablation in those with
or without PVC induced cardiomyopathy. PVC, premature ventricular
complex; LVEF, left ventricular ejection fraction; CMP, cardiomyopathy.
*P < 0.001