References
[1] Kondo M, Akachi S, Ando K, er al., “Two Japanese siblings
affected with Chikungunya fever with different clinical courses:
Imported infections from the Cook Islands”, J Dermatol , vol.43,
no. 6, pp697-700, 2016.
[2] Chow A, Her Z, Ong EK, et al., “Persistent arthralgia induced by
Chikungunya virus infection is associated with interleukin-6 and
granulocyte macrophage colony-stimulating factor”, J Infect Dis ,
vol.203, no.2, pp149-157, 2011.
[3] Liu X, Poo YS, Alves JC, et al., “Interleukin-17 Contributes to
Chikungunya Virus-Induced Disease”, mBio , vol.13, no.2,
e0028922, 2022.
[4] Boutet MA, Nerviani A, Lliso-Ribera G, et al., “Interleukin-36
family dysregulation drives joint inflammation and therapy response in
psoriatic arthritis”, Rheumatology (Oxford) , vol.59, no.4,
pp828-838, 2020.
[5] L. E. Jensen, “Interleukin-36 cytokines may overcome microbial
immune evasion strategies that inhibit interleukin-1 family signaling”,Sci Signal, vol.10, no.492, eaan3589, 2017.